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Survival
Factor in Neoplastic and Viral Diseases
By Dr. William
Koch (published 1961)
Go to original:
Survival Factor in Neoplastic and Viral Diseases
From Chapter Two.
VACCINE PROBLEMS
From what was stated so far it is seen that vaccines for a specific
virus do not immunize against the nucleoprotein that is the actual
pathogen, especially after it has penetrated and integrated with the
host cell, so to talk about curing cancer with vaccines or immune
sera is a waste of time. Even the prevention of viral infection by
vaccines is meeting the strongest statistical opposition since
large-scale smallpox and Salk vaccinations have been recorded. In
line with what is known about vaccine structure, statistics appear
logical when they show that paralytic “Polio” is increased both in
incidence and fatality by use of the vaccine. One may compare
various regions of different climatic conditions for the data. In
all of these the Salk Vaccine was enthusiastically applied, in
greater number each year, and the incidence increase was tremendous
each year, whereas, if the vaccine were effective there should have
been at least a little statistical improvement. In Montreal,
generally cool, they reported on August 27, 1959, 521 cases with 27
deaths, just while the “Polio” season was getting well under way, as
compared with less than one hundred in 1958. In Ottawa, generally
cool, 455 cases with 41 deaths were reported on August 22, 1959, as
compared with 64 cases with 7 deaths in 1958. In all of Canada, even
before the epidemic started to decline, there were 7 times more
paralytic cases in 1959 than in 1958, with a greater death rate. In
Detroit, much warmer, where vaccination was thorough, the number of
cases in 1958 was 697, against 226 in 1957. In the District of
Columbia, still warmer, the Health Department reported 7 times as
many cases in 1958 as in 1957. In New Jersey, in 1958, the Health
Department reported twice as great an incidence as in 1957. The
United States Public Health Service reported an increase of 15½% of
paralytic cases in 1958 over those in 1957 (49% against 33.5%). In
Hawaii (tropical) there were 65 victims including 32 paralytic cases
in 1958; half of these paralyzed cases (16) had received three Salk
shots, in an island where 60% had been vaccinated. In 1957 only 25
and 8/10ths % were paralytic instead of 49 and 9/l0ths% in 1958. If
the vaccine were effective there should have been a 60% decrease in
the incidence in the whole island of the paralytic infections,
instead of an increase of nearly 100%.
Nationwide statistics issued January 4, 1960, by the United States
Public Health Service, show that for the year 1959, up to December
26th (51 weeks), the increase in the incidence of Polio rose 85%
over that of the same period of 1958. There were 8,531 cases listed
for 1959, of which 5,661 were paralytic, as compared to 5,987 in
1958, of which 3,090 were paralytic. We just showed the great
increase in 1958 over the incidence of the total and the paralytic
cases of 1957. Where compulsory vaccination was practiced as in
North Carolina and Tennessee, Bealle’s investigations report a 400%
increase in paralytic and non-paralytic Polio during 1959 over 1958.
So it seems that the more vaccine that is used the more the actual
infection that comes about. The statistical analysis teaches much
about the nature of the virus.
Of course, this is comprehensible when one considers that the virus
breaks up into its component units on penetrating the host cell, as
if by a de-polymerization process, and it grows by acquiring new
units to add to each, as by a co-polymerization process. Some
investigators compare the viral structure to a deck of cards. The
complete deck or complete virus with all its units is the parent
pathogenic killer type. The vaccines may be regarded as incomplete
decks, with not all the units required to make up the full killer
type. Now, if a person carried vaccine units of, let us say, half or
less than the killer type requires and another vaccination or
infection by a crippled non-fatal virus comes along that presents
the units missing in the protective infection or vaccination of a
previous period either one of which alone can not produce the
disease, the units all added up could constitute the complete killer
type, and it has been shown that they are “shuffled” in at random to
make up the full virus, vaccination may add to the incidence of
serious or fatal infection, and the more the vaccination the more
the chance for building fatal viruses.
This happened in the writer’s early practice (1920). Two cases were
vaccinated against smallpox from the same vaccine lot. One had no
effect. The other came down with a rapidly fatal smallpox. There was
no epidemic at hand in Detroit at the time, so it was concluded that
the fatal case’s inoculation carried units required by a previous
silent infection to make it fatal.
SMALLPOX
Statistics on vaccination against Smallpox in the Philippines when
the United States took over are instructive. Reports run thus: In
1918, the Army forced the vaccination of 3,285,376 natives when no
epidemic was brewing, only the sporadic cases of the usual mild
nature. Of the vaccinated persons, 47,369 came down with Smallpox,
and of these 16,477 died. In 1919 the experiment was doubled.
7,670,252 natives were vaccinated. Of these 65,180 cases came down
with Smallpox, and 44,408 died. One sees here that the fatality rate
increased in the twice-vaccinated cases. In the first experiment,
one-third died, and in the second, two-thirds of the infected ones
died. This speaks for the retention of viral units from the previous
vaccinations, and indicates that, in the vaccine the shuffling in of
units varies in different specimens of vaccine. It should be stated
also that every epidemic of viral disease treated by the writer
followed vaccination within a few months, when protection should
have been had instead of an epidemic. This was so in Brazil, in
Aftosa, Cinemosa, Hog Cholera and Rabies, and in Cuba in Hog
Cholera.
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